On December 23, a research team from Riken and other institutes announced the results of a study showing that after infection with the novel coronavirus, the heart may be persistently infected with the virus and the risk of heart failure may increase, even if the patient does not develop noticeable heart disease. The team points to the possibility of a sharp increase in the number of heart failure patients in the near future and calls for countermeasures to be taken.

Infection with the novel coronavirus occurs when a protruding “spike protein” on the surface of the virus binds to the ACE2 receptor on the surface of human cells. According to the team, the heart is more likely to express ACE2 compared to other organs. It has also been reported that some people infected with the novel coronavirus have reduced cardiac function, but the detailed mechanism is not known.